The Laboratory for Calicivirus Studies

Peer Reviewed Publications




Appl Environ Microbiol 2000 Oct;66(10):4383-8 Related Articles, Books, LinkOut 

Concentration and detection of caliciviruses in water samples by reverse transcription-PCR.

Huang PW, Laborde D, Land VR, Matson DO, Smith AW, Jiang X.

Center for Pediatric Research, Children's Hospital of The King's Daughters

 and Eastern Virginia Medical School, Norfolk, Virginia 23510-1001, USA.

 

Human caliciviruses (HuCVs) cause waterborne outbreaks of gastroenteritis. Standard indicators of a safe water supply do not adequately predict contamination of water by viruses, including HuCVs. We developed a method to concentrate and detect HuCVs in water samples by using a cultivable primate calicivirus (Pan-1) as a model. Viable Pan-1 was seeded in different types of water and then filtered with a 1MDS filter, eluted with beef extract (BE), and reconcentrated by polyethylene glycol (PEG) precipitation. The viruses in the final samples were tested by plaque assay or by reverse transcription (RT)-PCR following extraction of the RNA with Trizol. Pan-1 was more sensitive to high-pH treatment than poliovirus was; a pH 9.0 BE solution was found to recover 35% more viable Pan-1 than a pH 9.5 BE solution recovered. Pan-1 was recovered from small volumes of deionized, finished, ground, and surface waters at efficiencies of 94, 73, 67, and 64%, respectively, when samples were assayed after elution without further concentration. When larger volumes of water (up to 40 liters) were tested after elution and concentration with PEG, 38, 19, and 14% of the seeded Pan-1 were recovered from finished, ground, and surface waters, respectively. The limit of detection of Pan-1 by RT-PCR was estimated to be 0.75 to 1.5 PFU in 40 liters of finished water. This method may be adapted for monitoring HuCVs in drinking water and other types of water for public health safety.

 

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Arch Virol 1999;144(1):199-208

Complete nucleotide sequence and genomic organization of a primate calicivirus, Pan-1.

Rinehart-Kim JE, Zhong WM, Jiang X, Smith AW, Matson DO.

Center for Pediatric Research, Children's Hospital of The King's Daughters, Norfolk, Virginia, USA.

 

The primate calicivirus, Pan-1, was originally isolated from several primate species. It displayed typical calicivirus morphology by electron microscopy. We determined the genomic sequence of Pan-1 by cDNA cloning and direct RNA sequencing. Pan-1 shares a similar genomic organization and a high degree of sequence identity with feline caliciviruses. The Pan-1 genome contains 8,304 nucleotides, plus a poly-A tail, and is longer than any other calicivirus strains with a completely known sequence. The extra sequences of Pan-1 include a unique 424-nucleotide sequence at the 5' end of ORF1, additional amino acids at the N-terminus of the capsid, and a longer 3' UTR.

 

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Arch Virol 1998;143(12):2421-30

Genomic mapping of a calicivirus VPg.

Dunham DM, Jiang X, Berke T, Smith AW, Matson DO.

Center for Pediatric Research, Children's Hospital of The King's Daughters Eastern Virginia Medical School, Norfolk, USA.

 

We identified a primate calicivirus (Pan-1) VPg in Pan-1-infected cells. The Pan-1 VPg was associated with both genomic and subgenomic RNAs. RNase digestion of Pan-1 RNA yielded a residual protein of 16 kDa. The N-terminal sequence of Pan-1 VPg was determined by direct amino acid sequencing and mapped to a region of the genome equivalent to picornavirus VPgs. Alignment of this protein sequence with similar regions of other calicivirus genomes allowed identification of conserved amino acid motifs and potential boundaries of the calicivirus VPg genes. Proteinase K treatment abolished the infectivity of Pan-1 RNA, suggesting that Pan-1 VPg is required for RNA infectivity.

 

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J Wildl Dis 1998 Jul;34(3):451-6

Isolation of reptilian calicivirus Crotalus type 1 from feral pinnipeds.

Barlough JE, Matson DO, Skilling DE, Berke T, Berry ES, Brown RF, Smith AW.

Laboratory for Calicivirus Studies, College of Veterinary Medicine, Oregon State University,

Corvallis 97331, USA.

 

Ten virus isolates were obtained from three species of marine mammals sampled on San Miguel Island (California, USA) and 1,200 km north on Rogue Reef (Oregon, USA) during tagging operations in 1986-87. Seven of these 10 were derived from 30 sampled Steller sea lion (Eumetopias jubatus pups, while two of 10 were isolated from one of 19 sampled California sea lion (Zalophus californianus californianus pups, and the remaining isolate was derived from 30 sampled northern fur seal (Callorhinus ursinus) pups. All 10 isolates were identified as belonging to a single serotype, reptilian calicivirus Crotalus type 1 (RCV Cro-1), previously isolated from both healthy and diseased snakes and frogs in a California zoologic collection. The marine samples also showed that nine of 30 Steller sea lion pups, one of 19 California sea lion pups and zero of 30 fur seal pups were producing type specific neutralizing antibodies to RCV Cro-1. This represents the first reported instance of the isolation from marine sources of calicivirus originally isolated from a terrestrial species.

 

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Emerg Infect Dis 1998 Apr;4(2):345-6

Reply to Drs. Capucci, Lavazza, and Mead.

Smith AW, Cherry NJ, Matson DO.

Oregon State University, Corvallis, Oregon, USA Neil J Cherry Lincoln University, Christchurch, New Zealand David O Matson Center for Pediatric Research, Norfolk,Virginia, USA.

 

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Clin Infect Dis 1998 Feb;26(2):434-9

In vitro isolation and characterization of a calicivirus causing a vesicular disease of the hands and feet.

Smith AW, Berry ES, Skilling DE, Barlough JE, Poet SE, Berke T, Mead J, Matson DO.

College of Veterinary Medicine, Laboratory for Calicivirus Studies, Oregon State University, Corvallis 97331, USA.

 

We report that a calicivirus of oceanic origin, San Miguel sea lion virus serotype 5 (SMSV-5), is a human pathogen. This biotype was isolated originally from blisters on the flippers of northern fur seals (Callorhinus ursinus) and replicates readily in primate and human cell lines. It infects a phylogenetically diverse array of hosts (poikilotherms to primates) and induces type-specific neutralizing antibodies in exposed humans. Group antibody against a pooled antigen of SMSV-5 and two other serotypes was also observed in 18% of 300 blood donors from a population in the northwestern United States. The human calicivirus isolate designated SMSV-5 Homosapien-1 (SMSV-5 Hom-1) was recovered from a laboratory worker with systemic illness, including vesicular lesions on all four extremities. We believe this newly described human disease represents a paradigmatic shift in calicivirus disease recognition.

 

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Emerg Infect Dis 1998 Jan-Mar;4(1):13-20

Comment in: Emerg Infect Dis. 1998 Apr-Jun;4(2):343-4; discussion 345-6

Comment in: Emerg Infect Dis. 1998 Apr-Jun;4(2):344-5; discussion 345-6

Calicivirus emergence from ocean reservoirs: zoonotic and interspecies movements.

Smith AW, Skilling DE, Cherry N, Mead JH, Matson DO.

College of Veterinary Medicine, Oregon State University, Corvallis 97331, USA. smithal@ccmail.orst.edu

 

Caliciviral infections in humans, among the most common causes of viral-induced vomiting and diarrhea, are caused by the Norwalk group of small round structured viruses, the Sapporo caliciviruses, and the hepatitis E agent. Human caliciviruses have been resistant to in vitro cultivation, and direct study of their origins and reservoirs outside infected humans or water and foods (such as shellfish contaminated with human sewage) has been difficult. Modes of transmission, other than direct fecal-oral routes, are not well understood. In contrast, animal viruses found in ocean reservoirs, which make up a second calicivirus group, can be cultivated in vitro. These viruses can emerge and infect terrestrial hosts, including humans. This article reviews the history of animal caliciviruses, their eventual recognition as zoonotic agents, and their potential usefulness as a predictive model for noncultivatable human and other animal caliciviruses (e.g., those seen in association with rabbit hemorrhagic disease).

 

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J Med Virol 1997 Aug;52(4):419-24

Phylogenetic analysis of the Caliciviruses.

Berke T, Golding B, Jiang X, Cubitt DW, Wolfaardt M, Smith AW, Matson DO.

Center for Pediatric Research, Children's Hospital of The King's Daughters,

Eastern Virginia Medical School, Norfolk 23510-1001, USA.

 

A phylogenetic portrait of the genus Calicivirus in the family Caliciviridae was developed based upon published sequences and newly characterized calicivirus (CV) strains, including additional Sapporo-like HuCV strains in pediatric diarrhea stool specimens from South Africa, the United Kingdom, and the United States. Distance and parsimony methods were applied to nucleotide and amino acid sequences of human and animal calicivirus 3D RNA-dependent RNA polymerase (approximately 470nt) and capsid hypervariable regions (approximately 1,200nt) to generate phylogenetic trees. Pairwise amino acid identity in the 3D region among the Sapporo-like strains ranged from 61% to 100%. Human and animal caliciviruses (HuCVs and AnCVs) separated into five genogroups: small round-structured viruses (SRSV), Sapporo-like, and hepatitis E virus (HEV)-like HuCVs and rabbit-, and vesicular exanthema of swine virus (VESV)-like AnCVs, each with a distinct genome organization. Each genogroup, including the Sapporo-like HuCVs, subdivided further into subgenogroups. The capsid region trees had higher levels of confidence than the 3D region trees and limited conclusions about genogroups could be drawn from the 3D region analyses. This analysis suggested that CVs include five potential virus subfamilies.

 

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Arch Virol 1996;141(12):2443-56

Partial characterization of the genome of nine animal caliciviruses.

Matson DO, Berke T, Dinulos MB, Poet E, Zhong WM, Dai XM, Jiang X, Golding B, Smith AW.

Center for Pediatric Research, Children's Hospital of The King's Daughters,

 Eastern Virginia Medical School, Norfolk, USA.

 

Caliciviruses (CVs) include at least 42 distinct serotypes. Seventeen CV serotypes have been isolated from marine sources and are called San Miguel sea lion caliciviruses (SMSVs). CVs also have been isolated from reptiles, primates, and other terrestrial animals. Nucleotide sequences from portions of genome of prototype strains for six SMSV serotypes, the reptile CV, Cro-1, the cetacean CV, Tur-1, and the primate CV, Pan-1, are presented. cDNA products of the polymerase (all strains characterized) and capsid (SMSV-17) regions were produced by reverse transcription-polymerase chain reaction using Pan-1 primers. Comparisons of nucleotide and amino acid identity among these and published CV sequences indicated that the nine characterized CVs fall into a phylogenetic group that includes SMSV-1 and SMSV-4 and that is more closely related to other characterized animal CVs than to most human CVs. The phylogenetic analysis also indicated that distinct genera exist among the Caliciviridae. SMSV-17 and SMSV-4 are predicted to be closer to each other than other caliciviruses of known serotype; 574 (82%) of the 704 amino acids in the SMSV-17 and SMSV-4 capsid genes were identical.

 

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Science 1996 Jul 5;273(5271):17-8

Comment on: Science. 1996 Apr 12;272(5259):191-2

Release of RHD virus in Australia.

Smith AW

 

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J Wildl Dis 1996 Jul;32(3):461-7

Detection of a non-cultivatable calicivirus from the white tern (Gygis alba rothschildi).

Poet SE, Skilling DE, Megyesl JL, Gilmartin WG, Smith AW.

College of Veterinary Medicine, Oregon State University Corvallis 97331, USA.

 

In April 1992, on Tern Island, French Frigate Shoals, Hawaii (USA), researchers observed a hand-reared white tern hatchling (Gygis alba rothschildi) develop vesicular lesions on the webbing between its toes, 6 days after falling out of its nest. Vesicular fluid collected from the foot lesions contained virus-like particles having typical calicivirus morphology. Calicivirus RNA was detected in the vesicular fluid by dot hybridization with a group-specific calicivirus copy DNA probe. Attempts to cultivate the virus in African green monkey kidney cells and porcine kidney cells were unsuccessful. This is the first report of a calicivirus infection associated with vesicular disease in a wild avian species.

 

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J Mol Biol 1994 Jul 15;240(3):256-64

Three-dimensional structure of calicivirus.

Prasad BV, Matson DO, Smith AW.

Verna and Marrs Mclean Department of Biochemistry, Baylor College of Medicine, Houston, TX 77030.

 

The Caliciviridae comprise a new family of single-stranded RNA viruses. While human caliciviruses cause gastroenteritis, the animal caliciviruses cause a wide range of diseases. We have determined the three-dimensional structure of a primate calicivirus using electron cryomicroscopy and computer image-processing techniques. Calicivirus is one of the rare animal viruses whose capsid is made of a single structural protein. The three-dimensional structure of the virus is distinct from that of any other animal virus. However, there are several architectural similarities with plant viruses such as tomato bushy stunt virus and turnip crinkle virus. The calicivirions are 405 A in diameter and exhibit T = 3 icosahedral symmetry. The main features of the three-dimensional structure are the 32 large surface hollows, 50 A deep and 90 A wide, at the icosahedral 5-fold and 3-fold axes, and the 90 distinctive arch-like capsomeres surrounding these hollows at the local and strict 2-fold axes. Each capsomere is a dimer of the capsid protein. Despite noticeable differences, the three quasi-equivalent subunits show common structural features: the upper bilobed domain, the central stem domain, and the lower shell domain. The 2-fold related capsid proteins interact through the bilobed domains to form the top of the arch. The structural differences between the connectors of the stem and the shell domain among the three subunits suggest the presence of a hinge region that may facilitate the capsid protein to adapt to the three quasi-equivalent environments of the T = 3 icosahedral structure. The shell domains of the pentavalent and hexavalent capsid proteins associate to form a continuous shell between the radii of 115 and 150 A. A beta-barrel structure has been suggested for the shell domain. The mass density in the inner shell between the radius of 85 and 110 A may contain a portion of the capsid protein interacting with the RNA. The features between the 45 and 85 A radius are suggestive of ordered RNA.

 

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Am J Vet Res 1990 Aug; 51(8):1184-7

New marine calicivirus serotype infective for swine.

Berry ES, Skilling DE, Barlough JE, Vedros NA, Gage LJ, Smith AW.

College of Veterinary Medicine, Oregon State University, Corvallis 97331.

 

A new serotype of calicivirus was isolated from California sea lions (Zalophus californianus) with severe vesicular disease. Neutralizing antibodies were found in 27 of 82 (32.9%) serum samples from California sea lions and in 15 of 146 (10.3%) serum samples from Steller sea lions (Eumetopias jubatus) tested. The seropositive animals were widely dispersed along the margins of the eastern Pacific basin, from the Bering Sea to the Santa Barbara Channel. Seropositive samples were found from as early as 1976 through the present time. This new calicivirus serotype, San Miguel sea lion virus type 13, was inoculated into weaned pigs, resulting in induction of severe vesicular disease, which spread to all pigs, including uninoculated pen contacts. Virus was continually shed by most of the pigs throughout the 2-week duration of the experiment.

 

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J Wildl Dis 1989 Jul;25(3):319-28

Mortality of harbor seal pups at different sites in the inland waters of Washington.

Steiger GH, Calambokidis J, Cubbage JC, Skilling DE, Smith AW, Gribble DH.

Cascadia Research Collective, Olympia, Washington 98501

 

We examined the mortality rates and causes of death of harbor seal (Phoca vitulina) pups in three regions of the inland waters of Washington (USA) in 1984. One hundred eight pups were collected during 239 searches of the shoreline areas near harbor seal haulout sites or through public reports. Minimum neonatal (up to 1 mo after birth) mortality rates at these regions ranged from 12% to 26% of the pups born. Neonatal mortality was highest in the Strait of Juan de Fuca; 33 of the estimated 105 (31%) pups born at the primary site died. Causes of death varied by location. In southern Puget Sound predation by coyotes (Canis latrans) was the primary cause of death, accounting for eight of 43 (19%) of the dead pups examined; starvation was the next most common cause of death. Mortality at study sites in the Strait of Juan de Fuca was related to premature parturition; 19 of 49 (39%) of the pups found dead were born prematurely. Nine species of bacteria were identified in samples taken from 42 pups; Proteus sp. and Escherichia coli were the most common.

 

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Am J Vet Res 1988 Dec;49(12):2018-21

Immunogenicity, pathogenicity, and transmissibility of a recombinant vaccinia virus in calves.

Wedman EE, Smith AW, Oliver RE.

College of Veterinary Medicine, Oregon State University, Corvallis 97331-4802.

 

Experiments concerned with the immunogenicity, pathogenicity, and transmissibility of a recombinant vaccinia:Sindbis virus were conducted. The WR strain of the recombinant vaccinia:Sindis virus was found to be infective for calves and mildly pathogenic, resulting in local tissue reaction. It was not transmissible to other calves. Also, it was found to be immunogenic when inoculated intradermally into calves, and antibody was produced against the parent vector virus (vaccinia) and the Sindbis antigen. Recombinant virus given IV to calves induced no detectable clinical signs, nor did the calves develop neutralizing antibodies. Furthermore, second-passage lesion material containing up to 10(7) tissue culture infective doses of the recombinant virus failed to induce development of lesions or illness in intradermally inoculated calves, and virus could not be recovered from the inoculation sites. In this series of experiments, this vaccinia recombinant given intradermally was immunogenic, mildly pathogenic at the local injection site only, and was not transmissible to contact animals, thus demonstrating the potential efficacy and safety of the WR strain of vaccinia virus when used as a live vector system in cattle.

 

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J Wildl Dis 1987 Oct;23(4):534-8

First isolation of a calicivirus from the Steller sea lion (Eumetopias jubatus).

Skilling DE, Barlough JE, Berry ES, Brown RF, Smith AW.

Calicivirus Research Laboratory, College of Veterinary Medicine, Oregon State University, Corvallis 97331.

 

A calicivirus was isolated from the rectum of a Steller sea lion (Eumetopias jubatus) pup on Rogue Reef, off the southern Oregon coast. Based on the results of neutralization tests with specific typing antisera, the isolate was identified as San Miguel sea lion virus serotype 6 (SMSV-6). Blood obtained from nine of 37 pups (24%) during virus sample collection procedures had specific neutralizing antibodies to SMSV-6. The isolation of SMSV-6 from a Steller sea lion represents, to our knowledge, the first isolation of any virus from this widely distributed marine mammal species, and serves to reconfirm the host-nonspecificity of yet another calicivirus of marine origin.

 

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J Wildl Dis 1987 Jan;23(1):45-51

Prevalence and distribution of serum neutralizing antibodies to Tillamook (bovine) calicivirus in selected populations of marine mammals.

Barlough JE, Berry ES, Smith AW, Skilling DE.

 

Neutralizing antibodies to Tillamook calicivirus (TCV) were found in sera collected from California sea lions (Zalophus c. californianus Lesson) in 1983 and 1984 and in sera collected from Steller sea lions (Eumetopias jubatus Schreber) in 1976 and 1985. The combined prevalence of antibodies for these two species was 10/228 = 4.38%. Titers ranged from 1:20 (five animals), to 1:40 (four animals), to 1:80 (one animal) by standard microtiter neutralization assay. The seropositive pinnipeds were dispersed widely along the margins of the eastern Pacific rim, from the Bering Sea to the Santa Barbara Channel. Antibodies to TCV were not found in sera collected from northern fur seals (Callorhinus ursinus L.), Pacific walruses (Odobenus rosmarus divergens Illiger), seals of the family Phocidae, or several cetacean species. Tillamook calicivirus was isolated originally in 1981 from dairy calves in Oregon; the finding of neutralizing antibodies in two widely distributed species of sea lions suggests the possibility of a marine origin for this agent.

 

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J Wildl Dis 1987 Jan;23(1):92-8

Serology and virology of the bowhead whale (Balaena mysticetus L.).

Smith AW, Skilling DE, Benirschke K, Albert TF, Barlough JE.

 

Sera from four bowhead whales (Balaena mysticetus L.) were examined for the presence of specific antibodies, and tissue and swab samples from six and four animals respectively were processed for isolation of viruses and for initiation of bowhead whale cell cultures. All sera were negative for antibodies to nine serovars of Leptospira interrogans and to 21 orthomyxovirus subtypes and a paramyxovirus (Newcastle disease virus). All sera were positive, however, for neutralizing antibodies to one or more calicivirus serotypes. Two untyped adenoviruses were isolated from colon samples of two different whales, but neutralizing antibodies to the agents could not be demonstrated in any sera. Three primary bowhead whale cell cultures were derived from kidney (two cultures) and testis (one culture), from three individual whales.

 

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J Wildl Dis 1987 Jan;23(1):34-44

Antibodies to marine caliciviruses in the Steller sea lion (Eumetopias jubatus Schreber).

Barlough JE, Berry ES, Goodwin EA, Brown RF, DeLong RL, Smith AW.

 

Sera from 145 Steller sea lions (76 adults, three subadults, 37 pups, and 29 fetuses) were tested for neutralizing antibodies to nine marine calicivirus serotypes. Antibodies were found to San Miguel sea lion virus (SMSV) types 1, 5, 6, 7, 8, 10 and 13, and to Tillamook (bovine) calicivirus, but no antibodies were found to the walrus calicivirus. Titers (microtiter neutralization assay) ranged from 1:20 to 1:320, with many positive reactions at the higher dilutions (greater than or equal to 1:80). Antibodies to SMSV's 5 and 10 were most common among animals sampled in Alaskan waters, while antibodies to SMSV-6 were most common among pups from the southern Oregon coast. These data provide evidence that Steller sea lions, like their California sea lion (Zalophus c. californianus Lesson) counterparts, have experienced widespread exposure to multiple serotypes of marine caliciviruses.

 

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Compendium of Continuing Education 1986; 8F5-F14

The marine calicivirus story (part 1)

Barlough JE, Berry ES, Skilling DE, Smith AW

 

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Compendium of Continuing Education 1986; F75-F82

The marine calicivirus story (part 2)

Barlough JE, Berry ES, Skilling DE, Smith AW

 

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Avian/Exotic Practice 1986; 3:8-20

Sea lions, caliciviruses, and the sea

Barlough JE, Berry ES, Skilling DE, Smith AW

 

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Diseases of Aquatic Organisms 1986; 2:73-80

Distribution in the North Pacific Ocean, Bearing Sea, and Arctic Ocean of animal populations known to carry a pathogenic calicivirus

Smith AW, Skilling DE, Barlough JE, Berry ES

 

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Journal of Clinical Microbiology 1986; 9:217-220

A simple method for the rapid preparation of virus isolates from cell culture material for electron microscopy

Skilling DE, Barlough JE, Berry ES, Smith AW

 

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Am J Vet Res 1986 Aug;47(8):1718-21

First isolation of calicivirus from reptiles and amphibians.

Smith AW, Anderson MP, Skilling DE, Barlough JE, Ensley PK.

 

Calicivirus isolations were made from 4 poikilothermic species in a zoologic collection. Viruses were recovered from 8 asymptomatic Aruba Island rattlesnakes (Crotalus unicolor; rectal swab samples) and from 8 symptomatic animals (4 Aruba Island rattlesnakes, 2 Bell's horned frogs [Ceratophrys orata], 1 rock rattlesnake [C lepidus], and 1 eyelash viper [Bothrops schlegeli] tissue samples obtained at necropsy). On the basis of cross-neutralization test results, the 16 isolates were antigenically indistinguishable and were considered to represent a unique calicivirus serotype, tentatively designated reptilian calicivirus Crotalus type 1. These isolations could not be associated causally with any specific disease entity either in naturally infected poikilotherms or in experimentally infected snakes and pigs.

 

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J Wildl Dis 1986 Apr;22(2):165-8

Antibodies to marine caliciviruses in the Pacific walrus (Odobenus rosmarus divergens Illiger).

Barlough JE, Berry ES, Skilling DE, Smith AW, Fay FH.

 

Sera from 155 Pacific walruses (Odobenus rosmarus divergens Illiger), sampled in the Chukchi Sea during the summer of 1983, were tested for serum neutralizing (SN) antibodies to six marine calicivirus serotypes. Serotypes tested included San Miguel sea lion virus (SMSV) types 1, 5, 8, and 10, previously isolated from northern fur seals (Callorhinus ursinus Linne) in the Bering Sea; walrus calicivirus (WCV), previously isolated from walrus feces collected off sea ice in the Chukchi Sea; and Tillamook calicivirus (TCV), a bovine isolate from Oregon of suspected marine origin. No antibodies were found to SMSV-1, SMSV-10, or TCV. Antibodies to SMSV-5 were found in two animals (titers 1:20 and 1:160); antibodies to SMSV-8 were found in four animals (all 1:20); and antibodies to WCV were found in one animal (titer 1:40). Antibodies to WCV have been found in the Pacific walrus previously; however, this represents the first report of antibodies to any of the SMSV serotypes in this marine mammal.

 

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Arch Virol 1985;84(3-4):181-95

Characterization of a new calicivirus isolated from feces of a dog.

Schaffer FL, Soergel ME, Black JW, Skilling DE, Smith AW, Cubitt WD.

 

Canine calicivirus (CaCV), isolated from feces of a dog with diarrhea, was readily propagated in cultures of canine cells and in a dolphin cell line. Serologic evidence indicated many dogs in at least one geographic area had been infected with CaCV, but its role as an etiologic agent of disease was not established. In cell culture most CaCV virions were strongly cell-associated making purification difficult. CaCV was established as a member of the Caliciviridae by morphology and physicochemical properties of virions (density, sedimentation rate, single major polypeptide, RNA genome size), although some of the properties differed slightly from those of previously described caliciviruses; evidence was also obtained for caliciviral RNA species in infected cells. Based on tests with antisera to numerous caliciviruses and presumed caliciviruses, CaCV appeared to be not closely related to any previously described virus except the stunting syndrome agent of chickens.

 

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J Wildl Dis 1985 Oct;21(4):426-8

Isolation of primate calicivirus Pan paniscus type 1 from a douc langur (Pygathrix nemaeus l.).

Smith AW, Skilling DE, Anderson MP, Benirschke K.

 

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Am J Vet Res 1985 Oct;46(10):2197-9

Calicivirus isolation from three species of primates: an incidental finding.

Smith AW, Skilling DE, Benirschke K.

 

Calicivirus isolations were made from 3 species of subhuman primates. Viruses were recovered from gingival lesions associated with periodontal disease in a spider monkey, from the oropharynx of a healthy silver leaf langur, and from the spleen of a lowland gorilla that had died of systemic coccidioidomycosis. Based on the results of cross-neutralization tests, all 3 isolates were serologically indistinguishable from a primate calicivirus Pan paniscus type 1. These isolations appeared to be incidental in nature and could not be associated causally with any specific disease entity.

 

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Res Vet Sci 1985 Jul;39(1):113-5

Immunisation of cattle with a recombinant togavirus-vaccinia virus strain.

Franke CA, Berry ES, Smith AW, Hruby DE.

 

Genetic engineering techniques have been used to construct a vaccinia virus recombinant which contains and expresses togavirus (Sindbis) genetic information. Intradermal inoculation of this recombinant strain into calves caused a transient pock-type lesion at the site of inoculation and elicited the production of substantial levels of anti-Sindbis virus neutralising antibodies. These results suggest that recombinant vaccinia virus vaccines may have potential for use in veterinary medicine.

 

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Am J Vet Res 1985 Jan;46(1):218-20

Isolation and identification of caliciviruses from dogs with enteric infections.

Evermann JF, McKeirnan AJ, Smith AW, Skilling DE, Ott RL.

 

Caliciviruses were isolated from 7 dogs and 1 captured coyote with enteritis. There was a high fatality rate in dogs 4 to 16 weeks of age. The occurrence in these dogs of concurrent infection with known enteric pathogens such as Salmonella sp, canine parvovirus, canine coronavirus, and canine rotavirus did not allow making any conclusions regarding the pathogenicity of this newly recognized calicivirus. The caliciviruses were characterized by electron microscopy and were further identified as being closely related to feline calicivirus by immunoelectron microscopy with specific antibody.

 

 

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Arch Virol 1983;76(3):257-61

Ultrastructure of newly recognized caliciviruses of the dog and mink.

Evermann JF, Smith AW, Skilling DE, McKeirnan AJ.

 

Two recently recognized viruses obtained from a dog with glossitis and from mink with hemorrhagic pneumonia were characterized by electron microscopy. The results of the negative-stained preparations indicated that the viruses were structurally compatible with the calicivirus group.

 

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J Am Vet Med Assoc 1983 Dec 1;183(11):1223-5

Calicivirus-induced vesicular disease in cetaceans and probable interspecies transmission.

Smith AW, Skilling DE, Ridgway S.

 

A calicivirus isolated from cetaceans is a new serotype designated cetacean calicivirus Tursiops-1 (CCV-Tur-1). It appears to have spread from an initially infected Atlantic bottlenose dolphin to a California sea lion, and was then carried by the sea lion to a second facility several miles away, where a second dolphin became infected and developed vesicular skin lesions that eroded, leaving shallow ulcers. Cetaceans and pinnipeds belong to separate orders, so this finding of interspecies transmission demonstrates the potentially broad host spectrum for yet another calicivirus.

 

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J Am Vet Med Assoc 1983 Dec 1;183(11):1219-22

Regression of cetacean tattoo lesions concurrent with conversion of precipitin antibody against a poxvirus.

Smith AW, Skilling DE, Ridgway SH, Fenner CA.

 

Tattoo lesions linked to the cetacean poxvirus of bottlenose dolphins regressed without treatment. Two types of regression were observed: (1) The tattoo lesions became raised and blanched, then disappeared along with sloughing skin. (2) When an incision was made through a tattoo lesion, the tattoo disappeared in a zone around the incision. Poxviruses removed from the raised, blanched skin lesions and from typical tattoo lesions were reacted with dolphin serums and examined by immunoelectron microscopy. Antibody was not detected against either of these poxvirus preparations when the dolphins had only typical tattoo lesions. However, after the raised, blanched lesions appeared, serums obtained during the acute or convalescent stages were positive for the poxvirus separated from the lesions. Regression of the typical tattoo lesions was concurrent with antibody conversion.

 

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Science 1983 Jul 1;221(4605):79-81

Calicivirus isolation and persistence in a pygmy chimpanzee (Pan paniscus).

Smith AW, Skilling DE, Ensley PK, Benirschke K, Lester TL.

 

What may be the first calicivirus isolate from any primate species, including man, was recovered from a herpesvirus-like lip lesion on a pygmy chimpanzee and then, 6 months later, from the throat of the same animal. The infected individual and its cage mates had circulating antibodies that were type-specific for this calicivirus. The agent was antigenically different from 30 other calicivirus serotypes and is tentatively designated primate calicivirus Pan paniscus type 1 (PCV-Pan 1).

 

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Am J Vet Res 1983 May;44(5):851-5

Isolation and partial characterization of a calicivirus from calves.

Smith AW, Mattson DE, Skilling DE, Schmitz JA.

 

A calicivirus was isolated from 3 dairy calves in a herd with persistent calf respiratory tract problems. This virus, named Tillamook calicivirus, was not neutralized by 18 different calicivirus-typing serums available. The agent caused only minimal lesions in 2 experimentally exposed calves, but did establish a persistent infection with virus shedding for 45 days, after which time the experiment was terminated. Experimentally exposed swine developed clinical vesicular lesions. The possible origins, disease potential, and relationships to the exotic animal disease agent, vesicular exanthema of swine are discussed for this first calicivirus isolate of bovine origin.

 

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J Wildl Dis 1983 Apr;19(2):86-9

New calicivirus isolates from feces of walrus (Odobenus rosmarus).

Smith AW, Ritter DG, Ray GC, Skilling DE, Wartzok D.

 

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Arch Virol 1981;67(2):165-8

Calcivirus (SMSV-5) infection in experimentally inoculated Opaleye fish (Girella nigricans).

Smith AW, Skilling DE, Prato CM, Bray HL.

 

At 15 degrees C, San Miguel sea lion virus infected fish (Girella nigricans), producing 10(7).6 TCID50 per gram of spleen, replicated in Vero cells (10(8) TCID50/gm) and retained viability after 14 days exposure to salt water (10(5) TCID50/ml dropped to 10(2).

 

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Am J Vet Res 1981 Apr;42(4):693-4

Isolation and identification of five new serotypes of calicivirus from marine mammals.

Smith AW, Skilling DE, Latham AB.

 

Five new serotypes of calicivirus have been isolated from marine mammals. San Miguel sea lion virus (SMSV)-8 and SMSV-10 were recovered from vesicular lesions on the flippers of northern fur seals in the Pribilof Islands of Alaska. Serotype SMSV-9 was isolated from a sea lion in southern California, and SMSV-11 was isolated from 2 northern fur seal pups in southern California. Serotype SMSV-12 was also isolated in southern California from sea lion and fur seal pups.

 

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Intervirology 1980;14(1):1-6

Caliciviridae.

Schaffer FL, Bachrach HL, Brown F, Gillespie JH, Burroughs JN, Madin SH, Madeley CR, Povey RC, Scott F, Smith AW, Studdert MJ.

 

The caliciviruses, as a proposed family Caliciviridae, have a distinct virion morphology with cup-shaped depressions on a spherical capsid surface. The viruses have single-stranded RNA, which has a molecular weight about 2.6 x 10(6) and is infectious. The RNA is covalently linked to a small protein. A single major polypeptide is found in the capsid. A subgenomic RNA, molecular weight about 1 x 10(6), coding for the capsid polypeptide is found in infected cells. Caliciviruses infecting swine, pinnipeds and cats have been characterized. Viruses which are morphologically identical to the known caliciviruses have been identified in human feces; these viruses have been shown to be associated with gastroenteritis, but they have not yet been propagated in the laboratory.

 

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Am J Vet Res 1980 Nov;41(11):1846-50

Preliminary investigation of a possible lung worm (Parafilaroides decorus), fish (Girella nigricans), and marine mammal (Callorhinus ursinus) cycle for San Miguel sea lion virus type 5.

Smith AW, Skilling DE, Brown RJ.

 

Colostrum-deprived neonatal Northern fur seal pups (Callorhinus ursinus) were exposed to San Miguel sea lion virus type 5 (SMSV-5) by feeding them fish (Girella nigricans) infected with virus or fish infected with both the sea lion lung worm larvae (Parafilaroides decorus) and virus. Virus infection was demonstrated in 8 of 9 pups, and 1 of these developed a vesicular lesion on the flipper. In this sequence, P decorus larvae exposed to SMSV-5 were fed to G nigricans held at 15 C in a salt water aquarium; 32 days later, these fish were killed, then fed to the fur seal pups. The vesicle developed 22 days subsequent to this and SMSV-5 was reisolated from the lesion. The SMSV-5 was shown to persist for at least 23 days in infected neonatal fur seals. Attempts to establish P decorus infection in Northern fur seal pups were apparently unsuccessful.

 

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Science 1980 Aug 22;209(4459):940-1

Calicivirus pathogenic for swine: a new serotype isolated from opaleye Girella nigricans,

an ocean fish.

Smith AW, Skilling DE, Dardiri AH, Latham AB.

 

A new calicivirus, designated San Miguel sea lion virus type 7 (SMSV-7), was isolated from fish and produced a disease condition identical to vesicular exanthema in experimentally infected swine. Serotype SMSV-7 was also isolated from four elephant seals and one sea lion trematode, whereas a second calicivirus serotype isolated from fish proved to be SMSV-6.

 

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J Wildl Dis 1980 Apr;16(2):261-6

Metastatic adenocarcinoma in two California sea lions, Zalophus c. californianus.

Brown RJ, Smith AW, Morejohn GV, DeLong RL.

 

Two California sea lions (Zalophus californianus californianus) came to necropsy with morphologically identical metastatic tumors. These were glandular epithelial origin and were widespread throughout the visceral organs. Both animals were found beached and dead within two months and were only 220 km apart.

 

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Arch Virol 1979;61(3):255-9

A new calicivirus isolated from a marine mammal.

Smith AW, Akers TG, Latham AB, Skilling DE, Bray HL.

 

A new serotype of calicivirus, designated as San Miguel sea lion virus type 6 (SMSV-6), was isolated from vesicular lesions on the flipper of a California sea lion pup. Serologic studies show that SMSV-6 neutralizing antibodies (SN) occur frequently among California sea lions and occasionally among northern fur seals. Feral swine, 1- to 6-week elephant seal pups and grey whales tested negative for SMSV-6 antibody.

 

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J Am Vet Med Assoc 1979 Nov 1;175(9):918-20

Viruses and virus diseases of marine mammals.

Smith AW, Skilling DE.

 

Poxvirus and several serotypes of calicivirus cause recognizable disease in marine mammals. Pox lesions in pinnipeds are raised and proliferative and are seen most frequently after confinement in captivity. In cetaceans, a poxvirus is associated with a much more benign and chronic lesion called a "tattoo." Numerous caliciviruses of differing antigenic types have been isolated from vesicular lesions and aborted fetuses of northern fur seals and California sea lions as well as from clinically normal and orphaned northern elephant seal pups. An adenovirus has been isolated from a sei whale and an enterovirus has been isolated from a gray whale.

 

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J Am Vet Med Assoc 1978 Nov 1;173(9):1131-3

Hazards of disease transfer from marine mammals to land mammals: review and recent findings.

Smith AW, Vedros NA, Akers TG, Gilmartin WG.

 

In a 5-year study (1972-1977) of microbial agents isolated from both clinically normal and diseased marine mammals, it was shown that certain disease agents are widespread in a diversity of ocean populations and that some are also transmissible to a number of terrestrial mammal species. Leptospira interrogans serovar pomona has been isolated repeatedly from 2 species of pinnipeds (Zalophus californianus califonianus and Callorhinus ursinus). Some of the more important bacterial pathogens for land mammals that were isolated from wild marine mammals are Pseudomonas mallei, Clostridium chauvoei, C novyi, Neisseria mucosa var heidelbergensis, Klebsiella pneumoniae, Salmonella spp, and Pasteurella multocida. Numerous serotypes of viruses classified as caliciviruses were isolated from a variety of marine mammals. Some of these are known to infect several land mammal species including swine horses, and primates. For this reason., precautions should be taken to ensure that disease agents shed by captive marine mammals are not transmitted to susceptible terrestrial mammals, including animal handlers and other human beings.

 

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Am J Vet Res 1978 Sep;39(9):1531-3

Immunoelectron microscopic comparisons of caliciviruses.

Smith AW, Skilling DE, Ritchie AE.

 

Using immunoelectron microscopy, 9 serotypes of vesicular exanthema of swine virus (VESV) were compared with 5 serotypes of San Miguel sea lion virus and 7 additional calicivirus isolates from marine animals. In addition, swine caliciviruses and marine caliciviruses were compared with the vaccinal strain of feline calicivirus (FCV) F-9. Of 9 VESV types, 8 showed common antigenicity with San Miguel sea lion virus. Of 9 VESV types, 2 showed common antigenicity with FCV F-9. All 12 marine caliciviruses showed common antigenicity with VESV, but not with FCV F-9

 

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Am J Vet Res 1978 Feb;39(2):291-6

Prevalence of vesicular exanthema of swine antibodies among feral mammals associated with the southern California coastal zones.

Smith AW, Latham AB.

 

Serum-neutralizing antibodies to both vesicular exanthema of swine virus (VESV) and San Miguel sea lion virus (SMSV) were found in a number of animal species having an association with the southern California coastal zones. California sea lions (Zalophus californianus) had antibodies to 9 VESV types (A48, C52, D53, E54, F54, G55, I55, J56, and K56). Fur seals (Callorhinus ursinus) and elephant seal pups (Mirounga angustirostris) were tested for antibodies to 6 VESV types and all were negative. California gray whales (Eschrichtius robustus) were tested for antibodies to 9 VESV types, and sperm whale (Physeter catodon), finback whale (Balaenoptera physalus), and sei whale (Balaenoptera borealis) were tested for 6 VESV types. Among the last 4 species, antibodies were present for each VESV type except C52, and all species were positive for antibodies to 2 or more VESV types. Feral swine from both Santa Cruz Island and Santa Catalina Island were tested and antibodies were present for 8 of 9 VESV types and all SMSV types except SMSV-4. One donkey from San Miguel Island was positive for VESV I55 and 2 were positive for SMSV-2.

 

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Am J Vet Res 1978 Feb;39(2):287-9

Caliciviruses infecting monkeys and possibly man.

Smith AW, Prato C, Skilling DE.

 

Caliciviruses have, for the 1st time, been shown experimentally to infect a primate. Twenty-four hours after being inoculated with San Miguel sea lion virus (SMSV), an African green monkey developed a febrile response and vesicular lesions at injection sites. Virus was recovered from lesion material 96 hours later and from the stool at 48 hours. Possible human infection with SMSV was indicated by serologic evidence. Three persons working with 4 distinct serotypes of SMSV developed neutralizing antibody titers to 2 SMSV types. The positive serum-neutralization test results were confirmed, using immunoelectron microscopy to demonstrate complexes of viruses and antibodies.

 

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Journal of Wildlife Diseases 1977;13:448-450

Calicivirus antibodies in wild fox populations

Prato CM, Ackers TG, Smith AW,

 

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Microbios Letters 1977;4:17-21

Premature Births in California sea lions: An overview

Smith AW, Skilling DE.

 

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Intervirology 1977;8(1):30-6

Characterization of two new serotypes of San Miguel sea lion virus.

Smith AW, Prato CM, Skilling DE.

 

Two new virus isolates, one from a California sea lion (Zalophus californianus californianus) and the other from a northern fur seal (Callorhinus ursinus) were partially characterized. Their physicochemical characteristics were similar to those of vesicular exanthema of swine virus (VESV) and San Miguel sea lion virus (SMSV). The virion morphology was in both instances typically calicivirus. On the basis of this and the serum cross-neutralization testing, these isolates were classed as two new types of SMSV and were designated serotypes SMSV-4 and SMSV-5.

 

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J Wildl Dis 1977 Apr;13(2):144-8

Naturally-occurring leptospirosis in northern fur seals (Callorhinus ursinus).

Smith AW, Brown RJ, Skilling DE, Bray HL, Keyes MC.

 

A 4-year study of Northern fur seal (Callorhinus ursinus) leptospirosis in the Bering Sea has shown that in newborn pups Leptospira pomona is associated with a multiple hemorrhage syndrome. Adults may develop an interstitial nephritis and shed organisms in the urine. The hed prevalence, based on microscopic slide agglutination tests, ranged between 7.0% and 15.4% for adult females and 3-4 year old bachelor bulls, whereas nursing pups averaging 4 months of age had a prevalence of 2%. These results are used to conclude that leptospirosis is not acquired primarily on the breeding rookeries but rather is more frequently acquired subsequent to the purps leaving the rookeries, presumably through the food chain during their first pelagic cycle.